CMAJ • September 13, 2005; 173 (6). doi:10.1503/cmaj.050011.
© 2005 CMA Media Inc. or its licensors
All editorial matter in CMAJ represents the opinions of the authors and not necessarily those of the Canadian Medical Association.
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Perioperative cardiac events in patients undergoing noncardiac surgery: a review of the magnitude of the problem, the pathophysiology of the events and methods to estimate and communicate risk

P.J. Devereaux, Lee Goldman, Deborah J. Cook, Ken Gilbert, Kate Leslie and Gordon H. Guyatt

From the Departments of Medicine and of Clinical Epidemiology and Biostatistics, McMaster University, Hamilton, Ont. (Devereaux, Cook, Guyatt), the Department of Medicine, University of California, San Francisco, San Francisco, Calif. (Goldman), the Department of Medicine, University of Western Ontario, London, Ont. (Gilbert), the Department of Anaesthesia and Pain Management, Royal Melbourne Hospital, and the Department of Pharmacology, University of Melbourne, Melbourne, Australia (Leslie)


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  		Fig. 1: Potential triggers of states associated with perioperative elevations in troponin levels, arterial thrombosis and fatal myocardial infarction. TNF-{alpha} = tumour necrosis factor-{alpha}, IL = interleukin, CRP = C-reactive protein, PAI-1 = plasminogen activator inhibitor-1, BP = blood pressure, HR = heart rate, FFAs = free fatty acids.

 

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